Download Advances in the Diagnosis and Treatment of Pancreatic by David C. Whitcomb MD PhD, Adam Slivka MD, Kenneth K. Lee PDF

By David C. Whitcomb MD PhD, Adam Slivka MD, Kenneth K. Lee

Issues of the pancreas have, long ago, been very complicated as the analysis used to be frequently made past due through the illness and no considerably helpful interventions have been to be had. this example is quickly altering as new insights from a number of views are built-in and serious about every one step of this advanced procedures. This factor of Gastroenterology Clinics of North the United States highlights a few parts of speedy growth in inflammatory and neoplastic problems of the pancreas. every one bankruptcy represents the built-in wisdom and viewpoint of specialists within the box, and characterize the hottest research of those state-of-the-art methods to complicated matters within the review and remedy of pancreatic issues.

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Extra info for Advances in the Diagnosis and Treatment of Pancreatic Diseases, An Issue of Gastroenterology Clinics Vol 36 Issue 2

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In severe cases, the fibroinflammatory process has no boundaries and can extend into the peripancreatic fat and can mimic superior mesenteric vein invasion by imaging studies. Such marked cases of fibrosis and inflammation can result in expansion of the involved area, resulting in a mass that can be described as an ‘‘inflammatory pseudotumor’’ or ‘‘tumefactive’’ AIP; mass lesions of up to 10 cm have been reported [7,61–64]. Another characteristic histologic feature of AIP is venulitis. Inflammatory cells not only surround small- and medium-sized veins, they also infiltrate the walls and the endothelium, which results in an obliterative venulitis (Fig.

Clinicopathological features of autoimmune pancreatitis in relation to elevation of serum IgG4. Pancreas 2005;31(1):28–31. [46] Hirano K, Shiratori Y, Komatsu Y, et al. Involvement of the biliary system in autoimmune pancreatitis: a follow-up study. Clin Gastroenterol Hepatol 2003;1(6):453–64. [47] Hirano K, Kawabe T, Yamamoto N, et al. Serum IgG4 concentrations in pancreatic and biliary diseases. Clin Chim Acta 2006;367(1–2):181–4. [48] Egawa N, Irie T, Tu Y, et al. A case of autoimmune pancreatitis with initially negative autoantibodies turning positive during the clinical course.

Autoimmune pancreatitis: radiologic findings in three histologically proven cases. J Comput Assist Tomogr 1998;22(6):880–3. [28] Irie H, Honda H, Baba S, et al. Autoimmune pancreatitis: CTand MR characteristics. AJR Am J Roentgenol 1998;170(5):1323–7. [29] Eerens I, Vanbeckevoort D, Vansteenbergen W, et al. Autoimmune pancreatitis associated with primary sclerosing cholangitis: MR imaging findings. Eur Radiol 2001;11(8):1401–4. [30] Sahani DV, Kalva SP, Farrell J, et al. Autoimmune pancreatitis: imaging features.

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